Breast Cancer Survivors

Chemo Secrets From a Breast Cancer Survivor

Undergoing chemotherapy can be one of the most terrifying things that you go through in your life. One of the most frightening things about chemotherapy is the lack of real information that most people have about it, and the unknown makes it so much more frightening as a result. This eBook, written by a young cancer survivor gives you the real story about what chemo is all about. The most valuable information you can get about chemotherapy is from someone that has already experienced it. This PDF eBook allows you to download and read it as soon as your order it. You can begin your journey of reassurance as soon as you want! Because that's what this is about: chemo does not have to be a terrifying unknown! Other people have gone through it before, and want to help you through it as well! This eBook is the guide through chemo that many people wish they could have had, and now you can have it yourself!

Chemo Secrets From a Breast Cancer Survivor Summary


4.6 stars out of 11 votes

Contents: Ebook
Author: Nalie Augustin
Price: $19.97

My Chemo Secrets From a Breast Cancer Survivor Review

Highly Recommended

All of the information that the author discovered has been compiled into a downloadable ebook so that purchasers of Chemo Secrets From a Breast Cancer can begin putting the methods it teaches to use as soon as possible.

As a whole, this manual contains everything you need to know about this subject. I would recommend it as a guide for beginners as well as experts and everyone in between.

Download Now

Kenneth H Mayer and HF Pizer

Advances in medical technology create new opportunities for nosocomial pathogens to proliferate in immunocompromised hosts and prosthetic tissues. Although secondary spread of these opportunistic infections is uncommon, nosocomial outbreaks of multi-resistant bacteria and fungi have plagued many intensive care units and cancer treatment centers. Advances in surgery and life support create new niches for microbes to proliferate, ranging from in-dwelling catheters to surgical wounds. As life-saving advances such as organ transplants, immunosuppressive therapy of autoimmune diseases like rheumatoid arthritis, and chemotherapy for malignancies become more accessible in under-resourced communities, the ability of a diverse array of microbes to create new clinical challenges will grow. There is no alternative to using antibiomicrobial drugs, and no single strategy for stopping the ability of micro-organisms to adapt to them. Hospitals can probably do a better job with infection control,...

Genetic Engineering and Society

A more immediate set of risks are the problems of confidentiality and discrimination associated with the newly developed capability to test for genetic predisposition to certain diseases. As mentioned previously, a gene has been discovered that predisposes women to breast cancer. Women who test positive for this gene can protect themselves by aggressive monitoring or by preemptive surgery. However, insurance companies may refuse to issue life or health insurance to women who possess this gene although not all will actually contract the disease. As a result, many women whose family history suggests that they might have the gene refuse to be tested.

Feeding behaviour and immunity

It appears that some sick insect larvae engage in a type of self-induced chemotherapy for example, larvae of Estigmene acrea sequester antiparasitic plant-derived pyrrolizidin alkaloids (Bernays and Singer, 2005). These authors demonstrated that taste sensitivity of parasitized caterpillars increases towards these antiparasitic compounds while at the same time they show decreased responsiveness to normally deterrent chemicals. This suggests that infected caterpillars might switch to food that would be unpalatable to an uninfected caterpillar with the purpose of eating more antiparasitic compounds.

The Low Dose Issue and InvertedU Dose Response Relationships for EDCs

For example, recent work by Welshons and colleagues examined effects in response to estradiol exposure in human breast cancer cells (Figure 2). Estradiol levels between 0.1 and 100 ppt produced an increased growth response in breast cancer cells, because at these levels, an increase in exposure causes an increase in the number of estrogen receptors bound by estradiol, thus leading to increased gene activation. At exposure levels in the typical toxicological dose range (part per million range), further increases in the dose of estradiol began to produce cell death. This result is extremely important for regulatory toxicology, because the high-level exposures in these experiments are analogous to those used for the prediction of risk posed by low doses, but the actual effects of low doses predicted to be safe have, until very recently, never been examined experimentally. Changes in dose within this very high part per million dose range cannot reveal variations in receptor-mediated gene...

Simulation of chemical reaction networks

The simulation of chemical reaction networks on SPICE has had significant applications. The methodology is rather simple (Wyatt 58 , Wyatt, Mikulecky and De Simone 59 ). The most extensive of these applications is in the area of biochemical pharmacological networks (Thakker, Wood, and Mikulecky 75 , Thakker and Mikulecky 76 , Walz, Caplan, Scriven, and Mikulecky 78 ). Let's look at an example from biology. This particular system, folate metabolism, is an important one in the synthesis of nucleic acids on the way to making building blocks for DNA and RNA. For that reason it plays an important role in cancer chemotherapy. (Seither, Trent, Mikulecky, Rape, and Goldman 71,72 , Seither, Hearne, Trent, Mikulecky, and Goldman 73 , White 79, 80 , White and Mikulecky 81 ).

The Achilles Heel of Malaria

The synergy here lies in the hypothesis that changes in odour profile may be linked to VOCs present in parasite-infected blood that are different from uninfected blood and that these VOCs may be exchanged with the lung cavity at the alveolar interface. In line with the development of breathalyzers for ailments in the developed world (lung cancer, breast cancer, early detection of heart transplant rejection, tuberculosis) we quickly realized the importance of the availability of such devices for non-invasive and rapid screening of patients for malaria parasites. The advantages are numerous (Knols 2005). In particular, a biomarker for gametocyte carriage would give tremendous power of tackling the infectious reservoir with gametocidal drugs or selective protection of hosts when carrying infectious stages.

Testing for Carcinogenicity and Teratogenicity

Rapid screening tests have been developed using especially sensitive strains of animals. Mouse skin is sensitive to tumor formation from topical application, apparently because it is active in biotransformation. Strain A mice spontaneously develop lung tumors in 100 of animals by 24 months of age. They can be used to determine carcinogenicity in tests as short as 12 to 24 weeks. Several rat strains have been developed with naturally high rates of breast cancer.

Issues Concerning Assessing Risks Posed by EDCs to Human Health

What triggers an investigation between an EDC and a human health effect Traditional ('classical') epidemio-logical studies were often designed to investigate unusual patterns of human health outcomes. Perhaps the most dramatic of these was the investigation of diethylstilbestrol (DES) in response to a cluster of seven cases of a rare vaginal cancer (clear cell adeno-carcinoma) in young women. Similarly, an awareness of increasing rates of lung cancer triggered the first studies of smoking and lung cancer. Some epidemiological studies of EDCs have similar origins. Indeed, DES itself is a quintessential EDC, and current research into possible EDC involvement in breast cancer causation and fertility impairment have been provoked by observations of human trends. All else being equal, the ability of an epidemiological study to identify the cause of an adverse outcome decreases as the prevalence of the outcome and the number of causal factors increase. For example, the identification of DES...

About the Editors

Miscarriage (Coping With A Miscarriage, with Christine O'Brien Palinski, The Dial Press, 1980), and artificial insemination (Having a Baby Without A Man, with Susan Robinson, MD, Simon & Schuster, 1985). He also co-authored Confronting Breast Cancer (with Sigmund Weitzman and Irene Kuter, Random House, 1987). From 1984 to 1994 he was founder and President of New England Medical Claims Analysts, a health-care consultancy, and during that time wrote and lectured on health-care cost containment. Presently he is Co-founder and Principal of Health Care Strategies, a consulting firm in Cambridge, Massachusetts, that provides program evaluation and management consulting services to community health-care providers, health-care systems, and social service organizations. Health Care Strategies specializes in working with clients to help them design and implement low-cost, practical systems for program evaluation. The goal is for providers and community-based organizations to be able to document...

Genetic Disease

Cancer is a result of a series of mutations (see Section 6.2.3). If some of these are inherited, a person is more susceptible to cancer, since fewer mutations remain to occur. For example, a gene associated with breast cancer has been discovered on chromosome 17 that is responsible for about 10 of all cases. Women with the mutation have an 80 to 90 chance of contracting the disease.

Theoretical Concepts

Altered gene expression and cellular signaling subsequent to development can cause transient changes, termed activational responses, or particularly through carcinogenesis, permanent detrimental effects. For example, lifetime exposure to estrogen is the best predictor of breast cancer in women, and exposure to EDCs that are 'environmental estrogens' could plausibly increase breast cancer risk. Thus, the impact of EDCs vary depending upon a variety of factors, including when in the life cycle of the organism exposure occurs, as well as the duration and amount of exposure. Until recently, the great importance of life stage, the very great vulnerability of the embryo, and the fact that consequences of fetal exposure could be entirely different from those seen from adult exposure had not been appreciated.


A variety of anthropogenic compounds found in nature have been shown to mimic the hormone estrogen xenoestrogens, endocrine disruptors, or environmental estrogens. These include the chlorinated pesticides atrazine, chlordane, DDT, endosulfan, kepone, and methoxychlor, as well as dioxins and some polychlorinated biphenyl (PCB) congeners. Several compounds associated with plastics are xenoestrogens as well Bisphenol A is released by polycarbonates when heated. Nonylphenol is a softener for plastics used in packaging and in flexible plastic tubing. Phthalates are also plastic softeners that are commonly used in food packaging and which have been found in laboratory experiments to cause reproductive disorders.The xenoestrogenic properties of some of these materials were discovered when laboratory investigations were confounded by their presense. In one case, cultures of breast cancer cells grew more rapidly than expected because of contamination from laboratory plasticware. Possible...